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Ap2 Vessels Quiz
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Multiple Choice Questions
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Edema
0%
the point where two blood vessels merge
0%
drives fluid out of capillary (major force)
0%
- by relaxation of the smooth muscl
0%
the accumulation of excess fluid in a tissue
Portal system
0%
Consists of veins on each side of the vertebral column that drain the back, thoracic and abdominal walls.
0%
wrap around capillaries and contract, regulating blood flow
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
2 consecutive capillary networks before returning to heart
Chemoreceptors are also located in the?
0%
medulla oblongata.
0%
aorta and carotid arteries.
0%
hyperproteinemia.
0%
considerably lower
Circulatory shock
0%
is any state in which cardiac output is insufficient to meet the body's metabolic needs.
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
The opposition to flow that blood encounters in vessels away from the heart
Aneurysm
0%
-Weak point in an artery or the heart wall-Most common sites: abdominal aorta, renal arteries, and arterial circle at the base of the brain
0%
is a quick and powerful way of altering blood pressure and flow
0%
wrap around capillaries and contract, regulating blood flow
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
Varicose veins
0%
supplies viscera and body wall
0%
failure of venous valves
0%
hyperproteinemia.
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
Most blood is in the
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
0%
veins
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
0%
The opposition to flow that blood encounters in vessels away from the heart
Distributing (muscular/medium) arteries
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
celiac trunk, superior mesenteric artery, renal arteries, gonadal arteries, inferior mesenteric artery, and common iliac arteries.
0%
- Increased capillary filtration • Kidney failure, histamine release, old age, poor venous return - Reduced capillary absorption • Hypoproteinemia, liver disease, dietary protein deficiency - Obstructed lymphatic drainage • Surgical removal of lymph node
0%
• Distributes blood to specific organs • Little to no elastic tissue • Brachial, femoral, renal, and splenic • Smooth muscle layers ¾ wall thicknes
Vasomotion
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
- by relaxation of the smooth muscl
0%
-chronic low resting BP -Caused by blood loss, dehydration, anemia
0%
is a quick and powerful way of altering blood pressure and flow
Interstitial hydrostatic pressure
0%
Arterial end = +3 out Venous end = +3 out
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = -8 outVenous end = -8 out
0%
Arterial end = 20 inVenous end = 20 in
drain blood from brain to
0%
internal jugular vein
0%
subclavian vein
0%
Arterial end = 20 inVenous end = 20 in
0%
aorta and carotid arteries.
Blood Pressure
0%
-Normal value, young adult: 120/75 mm Hg -Blood pressure (BP) - the force that blood exerts against a vessel wal-Sphygmomanometer and Brachial artery -Pulse pressure
0%
-Weak point in an artery or the heart wall-Most common sites: abdominal aorta, renal arteries, and arterial circle at the base of the brain
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
The fluids leave the capillaries at the arterial end because?
0%
celiac trunk, superior mesenteric artery, renal arteries, gonadal arteries, inferior mesenteric artery, and common iliac arteries.
0%
D. Central venous pressure being greater than pressure in the venules
0%
the net filtration pressure of the blood is higher at the arterial end than it is at the venous end.
0%
The opposition to flow that blood encounters in vessels away from the heart
The most important force driving reabsorption at the venous end of a capillary is
0%
blood colloid osmotic pressure.
0%
C. increased blood pressure
0%
draws fluid into capillary
0%
the great saphenous vein
Blood flow to the _______________ remains quite stable even when mean arterial pressure (MAP) fluctuates from 60 to 140 mm Hg.
0%
considerably lower
0%
hypothalamus
0%
the vagus nerve.
0%
the great saphenous vein
Three primary causes of Edema
0%
occurs when cardiac output is low because too little blood is returning to the heart.
0%
C. increased blood pressure
0%
- smallest arteries • Control amount of blood to various organs* - are most significant point of control over peripheral resistance and flow -Arterioles produce half of the total peripheral resistance
0%
- Increased capillary filtration • Kidney failure, histamine release, old age, poor venous return - Reduced capillary absorption • Hypoproteinemia, liver disease, dietary protein deficiency - Obstructed lymphatic drainage • Surgical removal of lymph node
Baroreceptors are located in the.?
0%
supplies 80% of the cerebrum
0%
aorta and carotid arteries.
0%
medulla oblongata.
0%
Arterial end = 30 out Venous end = 10 out
Exercise increases venous return in many ways:
0%
occurs when any object, such as a growing tumor or aneurysm, compresses a vein and impedes its blood flow.
0%
occurs when cardiac output is low because too little blood is returning to the heart.
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
Blood Hydrostatic pressures (BHP
0%
draws fluid into capillary
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = -8 outVenous end = -8 out
0%
drives fluid out of capillary (major force)
Hypotension
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
high blood pressure Chronic is resting BP > 140/90 Can weaken small arteries and cause aneurysms
0%
-chronic low resting BP -Caused by blood loss, dehydration, anemia
Venous pooling occurs with inactivity
0%
1. Local control 2. Neural control 3. Hormonal control
0%
- Venous pressure low- Prolonged standing - Jet pilot pressure suit
0%
the heart beats faster and harder, increasing cardiac output and blood pressure.
0%
D. Central venous pressure being greater than pressure in the venules
Arterial anastomosis
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
This is the longest vein, and portions of this vein are commonly used as grafts in coronary bypass surgery.
0%
the vagus and glossopharyngeal nerves.
0%
the vagus nerve.
0%
the great saphenous vein
0%
The first, second, and third choices are correct
Peripheral Resistance
0%
the heart beats faster and harder, increasing cardiac output and blood pressure.
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
The opposition to flow that blood encounters in vessels away from the heart
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
Neurogenic shock
0%
- Most common - One vein empties directly into another - Vein blockage less serious
0%
-chronic low resting BP -Caused by blood loss, dehydration, anemia
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
Vessel changes names as passes to different regions
0%
Arterial end = -8 outVenous end = -8 out
0%
Subclavian to axillary to brachial to radial and ulnar
0%
subclavian vein
0%
D. Central venous pressure being greater than pressure in the venules
Arteriovenous anastomosis (shunt)
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = 20 inVenous end = 20 in
0%
Arterial end = +3 out Venous end = +3 out
0%
Arterial end = -8 outVenous end = -8 out
Veins
0%
Greater capacity for blood than arterie
0%
receives most of the blood from the brain
0%
Arterial end = -8 outVenous end = -8 out
0%
wrap around capillaries and contract, regulating blood flow
Approximately what percent of fluid that exits the capillaries at the arterial end renters the capillaries at the venous end?
0%
90%
0%
tunica externa; vasa vasorum
0%
blood pressure.
0%
hypothalamus
Hypovolemic shock
0%
high blood pressure Chronic is resting BP > 140/90 Can weaken small arteries and cause aneurysms
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a form of venous pooling shock that results from a sudden loss of vasomotor tone, allowing the vessels to dilate.
0%
occurs when bacterial toxins trigger vasodilation and increased capillary permeability.
Net filtration or reabsorption pressure
0%
Arterial end = -8 outVenous end = -8 out
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = 13 outVenous end = 7 in
0%
Arterial end = 30 out Venous end = 10 out
Colloid osmotic pressures (COP) or oncotic pressure
0%
blood colloid osmotic pressure.
0%
drives fluid out of capillary (major force)
0%
Arterial end = 28 in Venous end = 28 in
0%
draws fluid into capillary
Internal carotid artery (ICA)
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = +3 out Venous end = +3 out
0%
supplies most external head structures
0%
supplies 80% of the cerebrum
Colloid osmotic pressures (COP) Tissue fluid
0%
aorta and carotid arteries.
0%
the vagus and glossopharyngeal nerves.
0%
drain the external structures of the head
0%
Arterial end = -8 outVenous end = -8 out
Oncotic pressure (net COP)
0%
Arterial end = -8 outVenous end = -8 out
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
Arterial end = +3 out Venous end = +3 out
0%
Arterial end = 20 inVenous end = 20 in
From superior to inferior, the major branches of the abdominal aorta are
0%
• Biggest arteries • Aorta, common carotid, subclavian, pulmonary trunk, and common iliac arteries • Much elastic tissue • Greatest pulse pressure
0%
-Normal value, young adult: 120/75 mm Hg -Blood pressure (BP) - the force that blood exerts against a vessel wal-Sphygmomanometer and Brachial artery -Pulse pressure
0%
- Raise or lower BP throughout the whole body - Re-routing blood from one region to another for perfusion of individual organs (Rest vs. Exercise
0%
celiac trunk, superior mesenteric artery, renal arteries, gonadal arteries, inferior mesenteric artery, and common iliac arteries.
Colloid osmotic pressures (COP) Blood
0%
the vagus and glossopharyngeal nerves.
0%
Artery flows directly into vein bypassing capillaries
0%
Arterial end = 28 in Venous end = 28 in
0%
Arterial end = 20 inVenous end = 20 in
Upper limb is drained by
0%
medulla oblongata.
0%
veins
0%
subclavian vein
0%
supplies 80% of the cerebrum
Systolic pressure
0%
peak arterial BP taken during ventricular contraction (ventricular systole)
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
link arterioles to capillaries
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
Arterioles
0%
- Raise or lower BP throughout the whole body - Re-routing blood from one region to another for perfusion of individual organs (Rest vs. Exercise
0%
- smallest arteries • Control amount of blood to various organs* - are most significant point of control over peripheral resistance and flow -Arterioles produce half of the total peripheral resistance
0%
• Biggest arteries • Aorta, common carotid, subclavian, pulmonary trunk, and common iliac arteries • Much elastic tissue • Greatest pulse pressure
0%
- Increased capillary filtration • Kidney failure, histamine release, old age, poor venous return - Reduced capillary absorption • Hypoproteinemia, liver disease, dietary protein deficiency - Obstructed lymphatic drainage • Surgical removal of lymph node
Internal jugular vein
0%
receives most of the blood from the brain
0%
supplies 80% of the cerebrum
0%
Arterial end = +3 out Venous end = +3 out
0%
supplies most external head structures
Caused by 3 factors of Peripheral Resistance are
0%
- Blood viscosity • Anemia and hypoproteinemia speed flow • Polycythemia and dehydration slow flow - Vessel length • The farther the liquid travels through a tube = more friction • Pressure and flow decline with distance - Vessel radius - most powerful influence over flow • Only significant way of controlling peripheral resistance. • Vasomotion
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
0%
draws fluid into capillary
0%
the net filtration pressure of the blood is higher at the arterial end than it is at the venous end.
Anaphylactic shock
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
occurs when bacterial toxins trigger vasodilation and increased capillary permeability.
0%
results from exposure to an antigen to which a person is allergic, such as bee venom.
0%
-Tissue necrosis, pulmonary and cerebral edema, circulatory shock
Metarterioles
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
0%
link arterioles to capillaries
0%
the point where two blood vessels merge
0%
failure of venous valves
Diastolic pressure
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
results from exposure to an antigen to which a person is allergic, such as bee venom.
0%
minimum arterial BP taken during ventricular relaxation (diastole) between heart beats
0%
- Two arteries merge - Collateral (alternative) routes of blood supply - Coronary circulation and around joints
Low Venous Return (LVR) Shock
0%
- Increased CO and BP - Increased respiratory rate (thoracic pump) - Increased skeletal muscle pump
0%
Artery flows directly into vein bypassing capillaries
0%
is caused by inadequate pumping by the heart, usually as a result of myocardial infarction.
0%
occurs when cardiac output is low because too little blood is returning to the heart.
- Vasodilation
0%
is a quick and powerful way of altering blood pressure and flow
0%
link arterioles to capillaries
0%
passes between clavicle and 1st rib
0%
- by relaxation of the smooth muscl
The action potential from the brain to the heart travels along...
0%
the vagus and glossopharyngeal nerves.
0%
subclavian vein
0%
the vagus nerve.
0%
blood colloid osmotic pressure.
Hypertension
0%
wrap around capillaries and contract, regulating blood flow
0%
is produced by a loss of blood volume as a result of hemorrhage, trauma, bleeding ulcers, burns, or dehydration.
0%
is a quick and powerful way of altering blood pressure and flow
0%
high blood pressure Chronic is resting BP > 140/90 Can weaken small arteries and cause aneurysms
Three ways of controlling vasomotion
0%
D. Central venous pressure being greater than pressure in the venules
0%
1. Local control 2. Neural control 3. Hormonal control
0%
Arterial end = 13 outVenous end = 7 in
0%
drain the external structures of the head
0 h : 0 m : 1 s
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